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Amenorrhea of ovarian origin

Its main feature, and that best differentiates primary ovarian failure secondary elevation of gonadotropins (low secondary).

This table appears to rule, after a variable period, fades away, giving a oligomenorrhea later ends in amenorrhea. There is a shortage of estrogen and increased gonadotropin. All this accompanied by the symptoms of menopause.

Because of this, there are odd references to the table as early menopause or premature ovarian failure. He starts talking about early menopause when this occurs before 35-40 years (there is no clear consensus).

The etiology of primary ovarian failure is unknown. Is classified according to whether or not follicular endowment.

Premature ovarian failure with follicular envelope

In this case, the ovaries contain many primordial follicles; this makes some doctors consider it a false early menopause. Gonadotropin levels are elevated. It includes:

• Syndrome Savage: Savage Syndrome or Syndrome of ovarian resistance to gonadotropins is characterized by the existence of primordial follicles, absence of developing follicles, with no in most cases, warning signs of autoimmune disease. The follicles are not sensitive to gonadotropins or endogenous (why are high) or exogenous.
• Autoimmune oophoritis, autoimmune oophoritis in the ovarian failure is due to the existence of circulating antibodies to granulosa cells, thecal antiovocitos or anticélulas. Developing follicles were observed, corpora lutea and atretic surrounded by lymphocytic infiltrate and plasma cells. Although 20% of cases are detected antiováricos antibodies, only 3% are associated with a syndrome that pluriglandular. Autoimmune diseases most frequently associated with autoimmune oofaritis are: Hashimoto's thyroiditis, Graves' disease, Addison's disease hypoparathyroidism, myasthenia gravis, diabetes mellitus, lupus erythematosus, idiopathic purpura trombocitoénica, pernicious anemia and hemolytic anemia among others.

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